Transregional Collaborative Research Centre 152
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Curriculum Vitae

Advanced Professional Positions

  • since 2017 Emmy Noether group leader, Institute of Clinical Neuroimmunology,
    LMU Munich
  • since 2017 Associated junior faculty member of the International Max Planck
    Research School for Molecular and Cellular Life Sciences (IMPRS-LS)

Professional Experience and Postdoctoral Training

  • 2014 – 2017 Project leader at Max Planck Institute of Neurobiology, Munich
    (Mentor: Prof. Hartmut Wekerle)
  • 2010 – 2014 Research fellow at Harvard Medical School and Brigham and Women's Hospital,
    Boston, USA, (Mentor: Prof. Vijay K. Kuchroo)

Academic Education

  • 2007 –2010 Ph.D. program at Ruhr-University Bochum, Germany and Center for Neurologic
    Diseases, Harvard Medical School, Boston, USA; ‘summa cum laude’
  • 2004 – 2006 Master of Science in Biochemistry at Ruhr-Universität Bochum and Center
    for Neurologic Diseases, Harvard Medical School, Boston, USA
  • 2001 – 2004 Bachelor of Science in Biochemistry at Ruhr-Universität Bochum

Honours, Awards, Scholarships and Other Qualifications

  • 2019 Certificate “Academic Teacher Training Course for Health Professions
  • 2017 Junior scientist award of the Sobek-Stiftung for Multiple Sclerosis Research
  • 2017 Emmy Noether grant, German Research Foundation (DFG)
  • 2012 Multiple Sclerosis Young Investigator Award from the Eva and Helmer Lehmann
    foundation
  • 2012 – 2013 Postdoctoral fellowship from the German Multiple Sclerosis society (DMSG)
  • 2007 – 2010 Boehringer Ingelheim Fonds Ph.D. scholarship
  • 2006 DAAD scholarship for conducting a master thesis project abroad
  • 2001 – 2003 Anniversary Scholarship by the German Chemical Industry Fund
    Freiburg
Professional Activities, Memberships
  • since 2020 PI of the DFG Collaborative Research Center 128 (“Initiating and effector versus
    regulatory mechanisms in multiple sclerosis”)
  • since 2018 Associated PI of the DFG Collaborative Research Center 1054 (“Control and
    Plasticity of Cell-Fate Decisions in the Immune System”

Publications

  • Gerdes LA, Yoon H, Peters A: Mikrobiota und Multiple Sklerose [Microbiota and multiple sclerosis]. Nervenarzt 91:1096-1107. German, 2020.
  • Matthias J, Heink S, Picard F, Zeiträg J, Kolz A, Chao YY, Soll D, de Almeida GP, Glasmacher E, Jacobsen ID, Riedel T, Peters A, Floess S, Huehn J, Baumjohann D, Huber M, Korn T, Zielinski CE: Salt generates antiinflammatory Th17 cells but amplifies pathogenicity in proinflammatory cytokine microenvironments. J Clin Invest 130:4587-4600, 2020.
  • Burkett PR*, Pawlak M*, Peters A*, Kuchroo VK: Chapter 6 „T cells and their subsets in autoimmunity“ in The Autoimmune Diseases, 6th edition, NR Rose, and IR Mackay, Eds. Academic Press. 91-116, 2019.
  • Mitsdoerffer M*, Peters A*: Tertiary Lymphoid Organs in Central Nervous System Autoimmunity. Front Immunol 7, 451, 2016.
  • Peters A*, Fowler KD*, Chalmin F, Merkler D, Kuchroo VK, Pot C: IL-27 induces Th17 differentiation in the absence of STAT1 signaling. J Immunol 195, 4144-53, 2015.
  • Peters A*, Burkett PR*, Sobel RA, Buckley CD, Watson SP, Bettelli E, Kuchroo VK: Podoplanin negatively regulates CD4+ effector T cell responses. J Clin Invest 125, 129-40, 2015.
  • Peters A, Pitcher LA, Sullivan JM, Mitsdoerffer M, Acton SE, Franz B, Wucherpfennig K, Turley S, Carroll MC, Sobel RA, Bettelli E, Kuchroo VK: Th17 cells induce ectopic lymphoid follicles in CNS tissue inflammation. Immunity 35, 986-96, 2011.
  • Jäger A, Dardalhon V, Sobel RA, Bettelli E, Kuchroo VK: Th1, Th17, and Th9 effector cells induce experimental autoimmune encephalomyelitis with different pathological phenotypes. J Immunol 183, 7169-77, 2009.
  • Awasthi A*, Riol-Blanco L*, Jäger A*, Korn T, Pot C, Galileos G, Bettelli E, Kuchroo VK, Oukka M: Cutting edge: IL-23 receptor gfp reporter mice reveal distinct populations of IL-17-producing cells. J Immunol 182, 5904-8, 2009.
  • Korn T*, Bettelli E*, Gao W*, Awasthi A, Jäger A, Strom TB, Oukka M, Kuchroo VK: IL-21 initiates an alternative pathway to induce proinflammatory T(H)17 cells. Nature 448, 484-7, 2007.
    *Equal contribution